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The American Journal of Pathology

The American Journal of Pathology

Vol. 185 Iss. 10, October 2015

Hepatic Expression of Serum Amyloid A1 Is Induced by Traumatic Brain Injury and Modulated by Telmisartan

Blocking angiotensin II receptor with telmisartan may be therapeutic for selected hepatic acute-phase response following traumatic brain injury.
MAGED1 deficiency results in osteopenia, decreased bone mineral density, and deteriorated bone architecture.
Identification of TMIGD1 may shed new light on the molecular mechanism of renal cell injury.
Prenatal use of the over-the-counter drug acetaminophen may have long lasting consequences.

Muscle-specific expression of certain exons results in different phenotypes in genetic muscular dystrophies.

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